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Update inference §1 (diagram)
Update inference §3 (diagram)
 
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{{#inference:id=1|to=Dexras1|tag=induces expression of}}<ref>{{cite web|url=https://www.nature.com/articles/srep28648|title=Dexras1 links glucocorticoids to insulin-like growth factor-1 signaling in adipogenesis|website=Nature|author=Hyo Jung Kim, Jiyoung Y. Cha, Jo Woon Seok, Yoonjeong Choi, Bo Kyung Yoon, Hyeonjin Choi, Jung Hwan Yu, Su Jin Song, Ara Kim, Hyemin Lee, Daeun Kim, Ji Yoon Han & Jae-woo Kim|quote=Previously, we showed that small G protein Dexras1 is expressed by glucocorticoids and leads to adipocyte differentiation}}</ref>
{{#inference:id=1|to=Dexras1|tag=induces expression of}}<ref>{{cite web|url=https://www.nature.com/articles/srep28648|title=Dexras1 links glucocorticoids to insulin-like growth factor-1 signaling in adipogenesis|website=Nature|author=Hyo Jung Kim, et al.|quote=Previously, we showed that small G protein Dexras1 is expressed by glucocorticoids and leads to adipocyte differentiation}}</ref>


== References ==
== References ==
<references />
<references />
{{#inference:id=3|to=CDK|tag=displaces ERα from|context=specifically CCND1/CDK2/CDK6, via FOXA1-primed chromatin sites}}<ref>{{cite web
|title=Glucocorticoid receptor modulation decreases ER-positive breast cancer cell proliferation and suppresses wild-type and mutant ER chromatin association.
|author=Tonsing-Carter E; Hernandez KM; Kim CR; Harkless RV; Oh A; Bowie KR; West-Szymanski DC; Betancourt-Ponce MA; Green BD; Lastra RR; Fleming GF; Chandarlapaty S; Conzen SD
|date=2019 Jul 24
|publisher=Breast cancer research : BCR
|url=https://pubmed.ncbi.nlm.nih.gov/31340854/
|pmid=31340854
|quote=
}}</ref>


[[Category:Receptor]]
[[Category:Receptor]]

Latest revision as of 22:57, 20 July 2026

induces expression of Dexras1[1]

References

  1. Hyo Jung Kim, et al.."Dexras1 links glucocorticoids to insulin-like growth factor-1 signaling in adipogenesis".
    Previously, we showed that small G protein Dexras1 is expressed by glucocorticoids and leads to adipocyte differentiation

displaces ERα from CDK (specifically CCND1/CDK2/CDK6, via FOXA1-primed chromatin sites)[1]

  1. Tonsing-Carter E; Hernandez KM; Kim CR; Harkless RV; Oh A; Bowie KR; West-Szymanski DC; Betancourt-Ponce MA; Green BD; Lastra RR; Fleming GF; Chandarlapaty S; Conzen SD."Glucocorticoid receptor modulation decreases ER-positive breast cancer cell proliferation and suppresses wild-type and mutant ER chromatin association.".