EGFR: Difference between revisions

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Update inference §2 (diagram)
Update inference §1 (diagram)
 
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{{#inference:id=1|to=Breast Ductal Lengthening|tag=facilitates}}<ref>{{cite web|url=https://doi.org/10.1073/pnas.0611647104|title=Amphiregulin is an essential mediator of estrogen receptor α function in mammary gland development|author=Laura Ciarloni; Sonia Mallepell; Cathrin Brisken|date=2007|quote=In fact, we find that estrogens induce amphiregulin through the ERα and require amphiregulin to induce proliferation of the mammary epithelium.}}</ref>
{{#inference:id=1|to=Breast Ductal Lengthening|tag=facilitates|inferred=yes}}<ref>{{cite web|url=https://doi.org/10.1073/pnas.0611647104|title=Amphiregulin is an essential mediator of estrogen receptor α function in mammary gland development|author=Laura Ciarloni; Sonia Mallepell; Cathrin Brisken|date=2007|quote=In fact, we find that estrogens induce amphiregulin through the ERα and require amphiregulin to induce proliferation of the mammary epithelium.}}</ref>


== References ==
== References ==

Latest revision as of 22:17, 20 July 2026

∴ → facilitates Breast Ductal Lengthening[1]

References

  1. Laura Ciarloni; Sonia Mallepell; Cathrin Brisken."Amphiregulin is an essential mediator of estrogen receptor α function in mammary gland development".
    In fact, we find that estrogens induce amphiregulin through the ERα and require amphiregulin to induce proliferation of the mammary epithelium.

activates PI3K (intracellular)